There is no western country in the world where there is no significant black white IQ gap.The gap is smallest at childhood but highest at adulthood. Again confirming the fact that genetic factors play the smallest during childhood while the biggest during adulthood.
(2013) Edward Dutton and Richard Lynn conclude that Finland has suffered a negative Flynn Effect between the years of 1997 and 2009 in all IQ metrics.
These results are in contrast to the gain Finland accrued between 1988 and 1997. The likely cause is the massively increased importation of migrants from other races, ethnicities, and cultures within that time period coupled with a slackening of mate selection criterion and racial endogamy. These findings are in-line with the consensus that outbreeding reduces IQ and the importation of migrants preempts that happening.
The manifestation of dysgenics in Finland supports IQ heritability, differences between races, and opposes the Flynn Effect and miscegenation propaganda in the same manner.
(2015) Michael Woodley and Curtis Dunkel conclude that France has suffered a 4-point drop in IQ scores in the decade between 1999 and 2008-9 during a period of mass migration from the Middle East and North Africa in particular.
Their conclusions are as follows
• Dutton and Lynn have identified a 4 point decline in French IQ. • Dysgenics and replacement migration are causes. • French IQ losses are found to be associated with the Jensen effect (ρ = .833). • A common factor of g loadings and 3 biological variables loads on the loss-vector. • This supports biological causation.
(2010) Douglas Massey concludes that human ethnocentrism is driven biologically and it is a natural, healthy phenomenon. This is indicated by increases in oxytocin when acting ethnocentrically and decreases when acting in a humanitarian manner to other races. He further concludes that oxytocin motivates in-group favoritism and derogation of the out-group.
(2012) Alvarez and Levy begin their study by stating their assumption that ethnic diversity will lead to gains in health for African and Mexican Americans and conclude their study by showing that it leads to a degradation in general health of African-Americans, an increase in health in White Hispanics, and a decrease in health in non-White Hispanics. The rates of heart disease and cancer are heightened for all races studied in more ethnically diverse communities.
(2012) Vogel, Monesson, and Scott conclude that infants develop ethnocentrism naturally as a means to help their survival and nurture their growth. Babies demonstrate this ethnocentrism before exposure to other races.
(2015) Dinesen et. al., conclude that being within 80 metres of an individual of another ethnicity or race reduces social trust and creates feelings of unease. This is concluded to be a natural evolutionary response to the out-group.
(1976) Sandra Scarr and Richard Weinberg conclude that Black children raised in wealthy White homes show better intellectual performance in their youth than Black children raised in Black homes. This supports the idea of youth malleability in the area of intelligence.
The White children in wealthy White homes performed even better in an amount matching the Black-White gap that can be seen in tests of IQ and academic performance outside of this experiment.
(1983) Sandra Scarr and Richard Weinberg conclude their experiment from the 1970s by testing the children again as adults.
Regardless of youth results, both races regressed to their respective racial means. The White-Black gap still existed and was not reduced.
These findings show that IQ malleability in youth exists, but it levels off as individuals age and subsequently regress to their racial means. These results further prove that there is a genetically heritable component to IQ and that there is an innate difference between different racial groups in this arena
(2015) Shakeshaft et al., analyze 360.000 sibling pairs, 9.000 twin pairs, and 3.000.000 18-year-olds in order to determine the heritability of IQ and its relation to genetic factors.
They conclude that IQ is, "familial, heritable, and caused by the same genetic and environmental factors responsible for the normal distribution of intelligence." They show that high-intelligence is, "a good candidate for, 'positive genetics,'" and conclude that there is a positive end to the distribution of intelligence as it is.
They show that there is no support for the Discontinuity Hypothesis that IQ is the result of environment or that mental illness is the result of something besides genetics.
In total, they conclude that IQ is extremely heritable (85% or more), genetic, polygenic, and does not arise from environmental factors but instead merely interacts with them. They show that people with exceptionally high IQs also tend to have other positive traits and very few negative genetic factors or expressed traits. They conclude IQ has almost no relation to de novo mutations, i.e., new mutations in DNA and that it nearly always matches what is heritable. They show that mental illness does not arise from environmental factors either and must arise from genetic factors and their interaction with the environment. These conclusions lend more credence to both Dual-Inheritance and the Continuity Hypothesis of IQs and Mental Illness.
(2009) Rushton & Ankney analyze data from various studies of over 1.000.000 individuals and confirm that there is a positive correlation between brain size and IQ. Larger skulls have larger brains on average and larger brains have higher IQs on average.
They conclude that this phenomenon is likely primarily genetic and as socioeconomic conditions reach a certain level the effects of nutrition on this narrow down and genetic gaps thus become more visible and exposed.
(2009) Rushton & Ankney analyze data from various studies of over 1.000.000 individuals and confirm that there is a positive correlation between brain size and IQ. Larger skulls have larger brains on average and larger brains have higher IQs on average.
They conclude that this phenomenon is likely primarily genetic and as socioeconomic conditions reach a certain level the effects of nutrition on this narrow down and genetic gaps thus become more visible and exposed.
(2005) Philippe Rushton and Arthur Jensen (author of, "The g Factor") conclude that IQ is the greatest indicator of future success in Western societies when inter-generational income dependence is accounted for.
They also found that IQ is at least 50% heritable and likely nearer to 80% heritable. To draw comparison, height is 70-90% heritable.
During their analysis they concluded that Whites have a minimum of 75% IQ heritability.
This means that crystallized and fluid forms of intelligence are both genetically transferrable from parent-to-child and have a sort of genetic, "stacking," effect.
The more intelligence alleles an individual possesses the greater expressed effect unlike epistatic genes like those for hair or skin color.
(2005) Toga & Thompson conclude that genetic influences on brain morphology also contribute significantly to IQ and in their own words, "A measure of cognitive ability, known as g, has been shown highly heritable across many studies. We argue that these genetic links are partly mediated by brain structure that is likewise under strong genetic control. Other factors, such as the environment, obviously play a role, but the predominant determinant appears to be genetic."
(2001) Thompson et al., conclude in their own words that, "A genetic continuum was detected in which brain structure was increasingly similar in subjects with increasing genetic affinity. Genetic factors significantly influenced cortical structure in Broca's and Wernicke's language areas, as well as frontal brain regions," and, "Preliminary correlations were performed suggesting that frontal gray matter differences may be linked to Spearman's g, which measures successful test performance across multiple cognitive domains (p < 0.05). These genetic brain maps reveal how genes determine individual differences, and may shed light on the heritability of cognitive and linguistic skills, as well as genetic liability for diseases that affect the human cortex."
This further suggests that brain structure, size, and cognitive performance are all heritable and genetic
